首页> 外文OA文献 >Mechanism of hypertriglyceridemia in human apolipoprotein (apo) CIII transgenic mice. Diminished very low density lipoprotein fractional catabolic rate associated with increased apo CIII and reduced apo E on the particles.

【2h】

Mechanism of hypertriglyceridemia in human apolipoprotein (apo) CIII transgenic mice. Diminished very low density lipoprotein fractional catabolic rate associated with increased apo CIII and reduced apo E on the particles.

机译：人载脂蛋白（apo）CIII转基因小鼠中高甘油三酯血症的机制。降低了极低密度脂蛋白分解代谢速率，与载脂蛋白CIII增加和颗粒上载脂蛋白E降低相关。

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

著录项
引文网络
相似文献
相关主题

著录项

作者
Aalto-Setälä, K; Fisher, E A; Chen, X; Chajek-Shaul, T; Hayek, T; Zechner, R; Walsh, A; Ramakrishnan, R; Ginsberg, H N; Breslow, J L;
展开▼
作者单位

展开▼
年度 1992
总页数
原文格式 PDF
正文语种
中图分类

相似文献

外文文献
中文文献
专利

1. Genetic study of common variants at the Apo E, Apo AI, Apo CIII, Apo B, lipoprotein lipase (LPL) and hepatic lipase (LIPC) genes and coronary artery disease (CAD): variation in LIPC gene associates with clinical outcomes in patients with established CAD [J] . Marco G Baroni, Andrea Berni, Stefano Romeo, BMC Medical Genetics . 2003,第1期

机译：Apo E，Apo AI，Apo CIII，Apo B，脂蛋白脂肪酶（LPL）和肝脂肪酶（LIPC）基因和冠状动脉疾病（CAD）常见变体的遗传研究：LIPC基因变异与以下患者的临床结局相关建立CAD
2. Increased levels of high-density lipoprotein cholesterol are ineffective in inhibiting the development of immune responses to oxidized low-density lipoprotein and atherosclerosis in transgenic rabbits expressing human apolipoprotein (apo) A-I with severe hypercholesterolaemia [J] . Agnès BOULLIER, Anne TAILLEUX, Catherine FIEVET, Clinical Science . 2001,第3期

机译：高密度脂蛋白胆固醇水平的升高无法有效抑制表达人载脂蛋白（apo）A-I的重度高胆固醇血症的转基因兔对氧化型低密度脂蛋白和动脉粥样硬化的免疫反应
3. Lycopene amends LPS induced oxidative stress and hypertriglyceridemia via modulating PCSK-9 expression and Apo-CIII mediated lipoprotein lipase activity [J] . Sahir Sultan Alvi, Irfan A. Ansari, Mohammad Kaleem Ahmad, Biomedicine & pharmacotherapy =: Biomedecine & pharmacotherapie . 2017,第期

机译：番茄红素通过调节PCSK-9表达和APO-CIII介导的脂蛋白脂酶活性来修复LPS诱导的氧化胁迫和高甘油三酯血症
4. Mapping free radical initiated oxidation of apo B-100 low density lipoprotein by LC-MS/MS [C] . Sourav Chakraborty, Matthew A. Tarr, Yang Cai American Society for Mass Spectrometry Conference on Mass Spectrometry and Allied Topics . 2010

机译：通过LC-MS / MS映射自由基引发APO B-100低密度脂蛋白的氧化
5. In vitro studies on the mechanisms responsible for the atherogenic nature of very low density lipoproteins in human subjects with type III or IV hyperlipoproteinemia and in apolipoprotein E deficient mice. [D] . Whitman, Stewart Charles. 1998

机译：在具有III型或IV型高脂蛋白血症的人类受试者和载脂蛋白E缺乏症小鼠中，导致极低密度脂蛋白的致动脉粥样硬化机理的机制的体外研究。
6. Mechanism of hypertriglyceridemia in human apolipoprotein (apo) CIII transgenic mice. Diminished very low density lipoprotein fractional catabolic rate associated with increased apo CIII and reduced apo E on the particles. [O] . K Aalto-Setälä, E A Fisher, X Chen, 1992

机译：人载脂蛋白（apo）CIII转基因小鼠中高甘油三酯血症的机制。降低了极低密度脂蛋白分解代谢速率与载脂蛋白CIII增加和颗粒上载脂蛋白E降低相关。
7. Dietary fat increases high density lipoprotein (HDL) levels both by increasing the transport rates and decreasing the fractional catabolic rates of HDL cholesterol ester and apolipoprotein (Apo) A-I. Presentation of a new animal model and mechanistic studies in human Apo A-I transgenic and control mice. [O] . Hayek, T, Ito, Y, Azrolan, N, 1993

机译：膳食脂肪通过增加HDL胆固醇酯和载脂蛋白（Apo）A-1的转运速率和降低分解代谢率来增加高密度脂蛋白（HDL）的水平。在人类Apo A-I转基因小鼠和对照小鼠中展示新的动物模型和机理研究。
8. Final Report Entitled Mechanism of the Effect of Different PFAS's (PFBS vs PFHS vs PFOS) on Lipid and Lipoprotein Metabolism in APO (Asterisks 3) Leiden/huCETP Transgenic Mice with Perfluorobutanesulfonate. [R] . 2010

机译：最终报告题为不同pFas（pFBs与pFHs与pFOs）对apO（asterisks 3）Leiden / huCETp全氟丁烷磺酸转基因小鼠脂质和脂蛋白代谢影响的机制。

Mechanism of hypertriglyceridemia in human apolipoprotein (apo) CIII transgenic mice. Diminished very low density lipoprotein fractional catabolic rate associated with increased apo CIII and reduced apo E on the particles.

著录项

引文网络

相似文献

相关主题

期刊订阅